PIPages
Labs

Andrew Ulijasz

University of Alabama at Birmingham, AL·Microbiology & Immunology

Everyday metabolism makes toxic waste. Among the most abundant of these byproducts is glyoxal, a highly reactive molecule produced mainly during glycolysis, the pathway that breaks down sugar.

Fields

Microbiology & Immunology

Biochemistry

Biophysics

Topics

glyoxal

dicarbonyl stress

advanced glycation end products

Grants
1 known grant, through Jan 2027

What they work on

  1. Map how GAD1, a heme-binding Antibiotic Monooxygenase domain protein, is regulated in Pseudomonas aeruginosa, including its co-transcription with GloA2, an enzyme that detoxifies glyoxal.
  2. Determine where GAD1 is distributed inside P. aeruginosa cells and identify its interactome, the set of partner proteins it binds, to test whether it redirects metabolism away from glycolysis.
  3. Solve the structures of GAD1 in its heme-free and heme-bound forms and in complex with interacting partners, to show how covalent glyoxal modification of the conserved residue Arg49 changes its behavior.

More about the research

Glyoxal chemically attacks specific amino acids in proteins, especially arginine, cysteine, histidine and lysine, degrading how those proteins work. In humans this dicarbonyl stress has been tied to cancer, diabetes, nervous system disorders, heart disease, hypertension, atherosclerosis and aging, and dedicated detoxifying systems exist across life because removing these molecules supports longevity. Even so, how a cell detects rising glyoxal and mounts a response is far less understood than oxidative stress.

Why it matters

Because this sensing pathway operates in the pathogen Pseudomonas aeruginosa, the genes and proteins uncovered could become new antimicrobial targets. GAD1-like proteins appear conserved up to humans, so the work may also clarify how human cells sense glyoxal buildup, an understudied stress linked to diabetes, cancer, heart disease and aging.

Known grants

From NIH and NSF research grants. Labs often have other funding we can't see.

A novel family of conserved glyoxal toxicity response proteins.

NIH · Feb 2022 – Jan 2027

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The school

University of Alabama at Birmingham: 45 labs on PI Pages, $866M a year on research, 166 research doctorates a year.

More about University of Alabama at Birmingham

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